Friday, June 22, 2012

P50 auditory sensory gating

P50 auditory sensory gating

Scholarly articles for p50 auditory sensory gating

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  1. Sensory gating - Wikipedia, the free encyclopedia - Similar
    Look up sensory gating in Wiktionary, the free dictionary. ... The P50 Auditory Gating deficit is one of the best established biological traits associated with ...

Sensory gating

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Sensory gating describes neurological processes of filtering out redundant or unnecessary stimuli in the brain from all possible environmental stimuli.[1][2] Also referred to as gating or filtering, sensory gating prevents an overload of irrelevant information in the higher cortical centers of the brain. The pulvinar nuclei of the thalamus play a major role in attention, and filter out unnecessary information.[3] Although sensory gating is largely automatic, it also occurs within the context of attentional processes. Though the term sensory gating has been used interchangeably with sensorimotor gating, the two are distinct constructs.[4]
  1. [PDF]

    Sensory Gating Measures Auditory P50 Response Prepulse Inhibition ...
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    Sensory Gating Measures. Auditory P50 Response. Prepulse Inhibition of Startle (PPI). Bruce Turetsky, M.D.. IOM Workshop. June 22, 2010 ...
  2. The adenosine antagonist theophylline impairs p50 auditory - NCBI
    by ES Ghisolfi - 2002 - Cited by 34 - Related articles
    The adenosine antagonist theophylline impairs p50 auditory sensory gating in normal subjects. Ghisolfi ES, Prokopiuk AS, Becker J, Ehlers JA, ...
  3. Yohimbine impairs P50 auditory sensory gating in normal subjects.
    by LE Adler - 1994 - Cited by 57 - Related articles
    Yohimbine impairs P50 auditory sensory gating in normal subjects. Adler LE, Hoffer L, Nagamoto HT, Waldo MC, Kisley MA, Giffith JM. University of Colorado ...
  4. [PDF]

    Auditory sensory gating deficit in abstinent chronic alcoholics
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    by J Marco - 2005 - Cited by 14 - Related articles
    Abstinent chronic alcoholics showed reduced P50 sensory gating. Present results suggest an inhibitory deficit in early pre-attentive auditory sensory processing ...
  5. Normal P50 Gating in Unmedicated Schizophrenia Outpatients
    by SM Arnfred - 2003 - Cited by 34 - Related articles
    Dec 1, 2003 – OBJECTIVE: The hypothesis of a sensory gating defect in schizophrenia has been supported by studies demonstrating deficient auditory P50...
  6. Caffeine modulates P50 auditory sensory gating in healthy subjects ...
    by ES Ghisolfi - 2006 - Cited by 15 - Related articles
    (2006) Ghisolfi et al. European neuropsychopharmacology the journal of the European College of Neuropsychopharmacology. Read by researchers in: 100% ...
  7. Reliability of P50 auditory sensory gating measures in infan ...

    by SK Hunter - 2008 - Cited by 5 - Related articles
    Jan 8, 2008 – This study assessed reliability of auditory sensory gating in young infants from 1-4 months of age u.
  8. Reduced P50 Auditory Sensory Gating Response in Professional ...
    by S Kizkin - 2006 - Cited by 9 - Related articles
    Click on any of the links below to perform a new search. Title: Reduced P50 Auditory Sensory Gating Response in Professional Musicians. Authors: Kizkin, Sibel ...
  9. [PDF]

    Comparison of sensory gating to mismatch negativity and self ...
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    by MA KISLEY - 2004 - Cited by 56 - Related articles
    Abnormal sensory gating of midlatency auditory. ERP components, particularly P1 (P50), has been interpreted to reflect the neural basis of stimulus filtering ...

Neuroimaging in Autism: Fractional anisotropy values - AJP CME Course for June 2012: Differences in White Matter Fiber Tract Development Present From 6 to 24 Months in Infants with Autism

Neuroimaging in Autism: Fractional anisotropy values - AJP CME Course for June 2012: Differences in White Matter Fiber Tract Development Present From 6 to 24 Months in Infants with Autism

fractional anisotropy values - GS 

normal fractional anisotropy values - GS 


Fractional anisotropy 

From Wikipedia, the free encyclopedia
Fractional anisotropy (FA) is a scalar value between zero and one that describes the degree of anisotropy of a diffusion process. A value of zero means that diffusion is isotropic, i.e. it is unrestricted (or equally restricted) in all directions. A value of one means that diffusion occurs only along one axis and is fully restricted along all other directions. FA is a measure often used in diffusion imaging where it is thought to reflect fiber density, axonal diameter, and myelination in white matter. The FA is an extension of the concept of eccentricity of conic sections in 3 dimensions, normalized to the unit range.


autism neuroimaging fractional anisotropy - Pubmed Search

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Autism neuroimaging - Fractional anisotropy - pubmed_result

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Diffusion tensor fractional anisotropy of the normal-appearing seven segments of the corpus callosum in healthy adults and relapsing-remitting multiple sclerosis patients

  1. Khader M. Hasan PhD1,*,
  2. Rakesh K. Gupta MD1,
  3. Rafael M. Santos MD, FRCS1,
  4. Jerry S. Wolinsky MD2,
  5. Ponnada A. Narayana PhD1
Article first published online: 19 MAY 2005
DOI: 10.1002/jmri.20296

Journal of Magnetic Resonance Imaging

Journal of Magnetic Resonance Imaging

Volume 21, Issue 6, pages 735–743, June 2005

How to Cite

Hasan, K. M., Gupta, R. K., Santos, R. M., Wolinsky, J. S. and Narayana, P. A. (2005), Diffusion tensor fractional anisotropy of the normal-appearing seven segments of the corpus callosum in healthy adults and relapsing-remitting multiple sclerosis patients. J. Magn. Reson. Imaging, 21: 735–743. doi: 10.1002/jmri.20296

Author Information

  1. 1 Department of Interventional and Diagnostic Imaging, University of Texas Medical School at Houston, Houston, Texas, USA
  2. 2 Department of Neurology, University of Texas Medical School at Houston, Houston, Texas, USA
*Department of Interventional and Diagnostic Imaging, University of Texas Medical School at Houston, 6431 Fannin Street, MSB 2.100, Houston, TX 77030
  1. Presented in part at the 12th Annual Meeting of ISMRM, Kyoto, Japan, 2004 (abstract 1498). The acquisition, processing, and quantitative analysis methodologies were also described in abstracts 338 and 1350 presented at the same meeting.

Publication History

  1. Issue published online: 19 MAY 2005
  2. Article first published online: 19 MAY 2005
  3. Manuscript Accepted: 7 FEB 2005
  4. Manuscript Received: 16 DEC 2004

Funded by

  • NIH. Grant Numbers: R01 NS31499, R01 EB02095
  • Dunn Research Foundation
  • Department of Diagnostic and Interventional Imaging, University of Texas at Houston




  • diffusion tensor imaging;
  • multiple sclerosis;
  • corpus callosum



To investigate the utility of whole-brain diffusion tensor imaging (DTI) in elucidating the pathogenesis of multiple sclerosis (MS) using the normal-appearing white matter (NAWM) of the corpus callosum (CC) as a marker of occult disease activity.

Materials and Methods

A high signal-to-noise ratio (SNR) and optimized entire brain DTI data were acquired in 26 clinically-definite relapsing and remitting multiple sclerosis (RRMS) patients and 32 age-matched healthy adult controls. The fractional anisotropy (FA) values of seven functionally distinct regions in the normal-appearing CC were compared between patients and controls.


This study indicates that 1) there was a gender-independent FA heterogeneity of the functionally specialized CC segments in normal volunteers; 2) FA in the MS group was significantly decreased in the anterior (P = 0.0039) and posterior (P = 0.0018) midbody subdivisions of the CC, possibly due to a reduction of small-caliber axons; and 3) the FA of the genu of the CC was relatively intact in the MS patients compared to the healthy age-matched controls (P = 0.644), while the splenium showed an insignificant trend of reduced FA values (P = 0.248). The decrease in FA in any of the CC subdivisions did not correlate with disease duration (DD) or the expanded disability status scale (EDSS) score.


The preliminary results are consistent with published histopathology and clinical studies on MS, but not with some published DTI reports. This study provides insights into the pathogenesis of MS, and the role played by compromised axonal integrity in this disease. J. Magn. Reson. Imaging 2005;21:735–743. © 2005 Wiley-Liss, Inc.

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June 01, 2012
AJP CME Course for June 2012: Differences in White Matter Fiber Tract Development Present From 6 to 24 Months in Infants with Autism
Expires May 31, 2014

Estimates of the fractional anisotropy slope parameters with standard errors are presented for the ASD-positive and -negative groups for all tracts in Table 2. Both groups showed significant increases in fractional anisotropy from 6 to 24 months, though the rate of change for the ASD-negative group was significantly greater than that for the ASD-positive group in the bilateral limbic (fornix) and association (inferior longitudinal fasciculus and uncinate) fiber tracts. Individual and mean group trajectories for these tracts are presented in Figure 1. The changes from 6 to 24 months in fractional anisotropy for the corpus callosum subdivisions are shown in Figure 2; the change for the body was significantly different in the two groups. For projection tracts, the growth trajectories of the left anterior thalamic radiation and all internal capsule divisions were significantly steeper for the ASD-negative infants (Figure 3).

June 01, 2012
AJP CME Course for June 2012: Differences in White Matter Fiber Tract Development Present From 6 to 24 Months in Infants with Autism
Expires May 31, 2014
Fractional anisotropy values may be generated for white matter fiber tracts. Values in the high range (e.g., 0.8–1.0) are indicative of what quality?

A. Isotropic diffusion

B. Transverse diffusion

C. Weak directional diffusion

D. Strong directional diffusion

At 6 months old, cross-sectional fractional anisotropy values for autism spectrum disorder (ASD)-negative and ASD-positive groups differed for which of the following white matter tracts?

A. Right uncinate fasciculus

B. Left inferior longitudinal fasciculus

C. Left anterior thalamic radiation

D. Splenium of corpus callosum

In typical white matter development during infancy, what two processes combine to ensure efficient structural connectivity between brain regions?

A. Axon pruning and myelination

B. Apoptosis and glial cell proliferation

C. Neural refinement and canalization

D. Microglial activation and synaptogenesis


A Critique of the DSM-5 Field Trials - Journal of Nervous & Mental Disease: Original Article

Journal of Nervous & Mental Disease:
June 2012 - Volume 200 - Issue 6 - p 517–519
doi: 10.1097/NMD.0b013e318257c699
Original Article

A Critique of the DSM-5 Field Trials

Jones, K. Dayle PhD, LMHC

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Abstract: This article provides an overview and critique of the field trials for the current revision of the Diagnostic and Statistical Manual of Mental Disorders, Fifth Edition (DSM-5). The purpose of the DSM-5 field trials was to evaluate the use, feasibility, safety, reliability, and validity of the DSM-5 proposals. In this article, the procedures for evaluating these properties of the DSM-5 are reviewed, and several concerns—such as delays, disorganization, missed deadlines, field trial cancelations, lack of adequate validity testing, and high clinician attrition rates—and their likely impact on the field trial results are presented.
© 2012 Lippincott Williams & Wilkins, Inc.

Defense Rebuts Insanity Claim in Breivik Trial - ABC News

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Defense Rebuts Insanity Claim in Breivik Trial
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Defense Rebuts Insanity Claim in Breivik Trial. ... him legally sane, saying he suffers from a dissocial and narcissistic personality disorder, but is not psychotic.
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Defense Rebuts Insanity Claim in Breivik Trial

On the last day of his trial, Anders Behring Breivik's defense lawyers on Friday tried to cast the confessed mass killer as a political militant motivated by an extreme right-wing ideology rather than a delusional madman who killed 77 people for the sake of killing.
Since Breivik has admitted to the bomb-and-gun attacks on July 22, the self-styled anti-Muslim militant's mental state has been the key focus of the 10-week trial.
Nevertheless, Breivik's defense lawyer Geir Lippestad requested that the 32-year-old Norwegian be acquitted or given the mildest possible prison term for the country's worst peacetime massacre. The plea for acquittal was made out of principle, without any realistic chance of success: Breivik claims he acted in defense of his nation and that the killings were therefore justified.
In his closing arguments, Lippestad reiterated that Breivik accepts that he set off a bomb outside a government high-rise and then gunned down dozens of teenagers at a Labor Party youth camp in the way that the attacks were described in court.
"That little, safe Norway would be hit by such a terror attack is almost impossible to understand," Lippestad said. And that helps explain why psychiatric experts reached different conclusions about Breivik's mental state, he added.
Lippestad tried to prove to the court that Breivik's claims of being a resistance fighter in a struggle to protect Norway and Europe from being colonized by Muslims are not delusional, but part of a political view shared by other right-wing extremists.
Anders Behring Breivik, the confessed gunman... View Full Caption

He also refuted assertions by one team of psychiatrists that the driving force behind Breivik's attacks was a psychotic impulse to kill, rather than a political ideology.
"July 22 was an inferno of violence," Lippestad said. "But we must also look at how he carried out the attacks to see whether it was violence in itself or radical politics that was the cause."
"He realized that it is wrong to kill but he chose to kill. That's what terrorists do," Lippestad said. "The ends justify the means. You don't understand this if you don't understand the culture of right-wing extremists."
When Breivik talks about a civil war he's not fantasizing about tanks and soldiers in the forest, but referring to a low-intensity struggle he believes will last for 60 years, Lippestad said.
"None of us know what Europe will look like in 60 years," Lippestad said. "Who would have thought 10 years ago that a right-wing extremist party in Greece would get 10 percent in the election now?"
Two teams of psychiatrists reached opposite conclusions about Breivik's mental health. The first team diagnosed him with "paranoid schizophrenia," a serious mental illness. The second team found him legally sane, saying he suffers from a dissocial and narcissistic personality disorder, but is not psychotic.
Prosecutors on Thursday called for an insanity ruling, saying there was enough doubt about Breivik's mental state to preclude a prison sentence.
The five-judge panel is expected to make a ruling in July or August. If deemed mentally competent, Breivik would likely be given Norway's maximum prison term of 21 years. A sentence can be extended beyond that if a prisoner is considered a menace to society. If declared insane, he would be committed to a mental institution for as long as he's considered sick and dangerous to others. Prosecutors suggested Thursday that could mean he would be held for the rest of his life.
Lippestad's otherwise focused statement ended on a confusing note when he asked the court for the most lenient possible prison sentence for his client. After being corrected by Breivik, Lippestad said the defense asks for an acquittal or a lenient sentence, but primarily wants the court to reject the insanity claim.

Prosecutors want mass killer Breivik ruled insane - Reuters

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via Breivik Syndrome - Google Blog Search by Baron Bodissey on 6/21/12
This concerns a very nutty Norwegian professor who sincerely hopes that Anders Behring Breivik will be given the chance to return to society at some stage. Notice also ..... Down's Syndrome Babies, Sarah Palin and the BBC ...

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The traumatic trial of confessed Norwegian terrorist Anders Behring Breivik has raised major questions over the role of forensic psychiatry in Norway's legal system. Conflicting diagnoses of Breivik's mental health have resulted in serious doubt over whether he can be punished for his ... and News from Norway/Nina Berglund. Please support our news service. Readers in Norway can use our donor account. Our international readers can click on our “Donate” button: ...